Oncogene and transgenics correlates of cancer risk assessments
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Oncogene and transgenics correlates of cancer risk assessments

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Published by Plenum Press in New York .
Written in English

Subjects:

  • Oncogenes -- Congresses,
  • Cancer -- Risk factors -- Congresses,
  • Health risk assessment -- Congresses,
  • Transgenic animals -- Congresses,
  • Cancer -- Animal models -- Congresses,
  • Mice, Transgenic -- genetics -- congresses,
  • Neoplasms -- genetics -- congresses,
  • Oncogenes -- congresses,
  • Risk Factors -- congresses

Book details:

Edition Notes

Statementedited by Constantine Zervos.
SeriesNATO ASI series., v. 232
ContributionsZervos, Constantine., North Atlantic Treaty Organization. Scientific Affairs Division., NATO Advanced Research Workshop on Oncogene and Transgenics Correlates of Cancer Risk Assessments (1991 : Attikē, Greece)
Classifications
LC ClassificationsRC268.42 .O5 1992
The Physical Object
Paginationxi, 351 p. :
Number of Pages351
ID Numbers
Open LibraryOL1718993M
ISBN 100306442426
LC Control Number92021812

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Oncogene and Transgenics Correlates of Cancer Risk Assessments (Nato Science Series A:): Medicine & Health Science Books @ ISBN: OCLC Number: Notes: "Proceedings of a NATO Advanced Research Workshop on Oncogene and Transgenics Correlates of Cancer Risk Assessments, held October , , in Vouliagmeni Beach, Attiki, Greece"--Title page verso.   Editorial Reviews. A product of the NATO Advanced Research Workshop on [title], held in Attiki, Greece, October , this volume advances the state of the art of cancer risk assessment methods by identifying potential short and long term contributions to such methods from the fundamental biological disciplines, i.e. biochemistry, cell biology, molecular biology, molecular genetics of cancer Pages: ISBN: OCLC Number: Description: 1 online resource ( pages) Contents: Outstanding Problems in Cancer Risk Assessments: Pharmacodynamic Models for Cancer Risk Assessment; S.H. Moolgavkar.

Anderson E.L. () Cancer Risk Assessments: An Overview. In: Zervos C. (eds) Oncogene and Transgenics Correlates of Cancer Risk Assessments. NATO ASI Author: Elizabeth L. Anderson.   Buy Oncogene and Transgenics Correlates of Cancer Risk Assessments by Constantine Zervos from Waterstones today! Click and Collect from your local Waterstones or get FREE UK delivery on orders over £Book Edition: Softcover Reprint of The Original 1st Ed. 1. Author(s): Zervos,Constantine; North Atlantic Treaty Organization. Scientific Affairs Division.; NATO Advanced Research Workshop on Oncogene and Transgenics Correlates of Cancer Risk Assessments,( Attikē, Greece) Title(s): Oncogene and transgenics correlates of cancer risk assessments/ edited by Constantine Zervos. Book review Full text access Oncogene and Transgenics Correlates of Cancer Risk Assessments, Constantine Zervos (Ed.). Plenum Press, New York and London (), pages, published in cooperation with NATO Scientific Affairs Division, NATO ASI Series A: Life Sciences., ISBN: 0 .

In: Oncogene and transgenics: Correlates of cancer risk assessments. C Zervos (Ed). Plenum Publishers, New York, pp, Haliassos A and SPANDIDOS DA: K-ras oncogene activation in neoplasias of patients with familial adenomatous polyposis or Kidney transplant. In: Oncogenes and transgenics: Correlates of Cancer risk assessments. Cancer genetics risk assessment and genetic counseling includes family history, psychosocial assessments, and education on hereditary cancer syndromes, testing, and risk. Get more information including the ethical, legal, and social implications of genetic testing in this summary for clinicians. Inherited mutations increase a person's risk of developing cancer at an earlier age because they already have a mutation in a gene that is known to control the cell cycle in some way. Eventually, more mutations will happen and add to the problem.   The nature of the cancer cell-of-origin. This cancer cell-of-origin (or cancer-initiating cell, not to be confused with the CSCs which, as we have explained, are the cancer-maintaining cells of a tumour that is already developed) is the, initially, healthy cell (it doesn't necessarily have to be a stem cell) that will be reprogrammed by the oncogenic hit(s) and will finally give rise to a (pre Cited by: